Cardarine (GW501516): Benefits, Cancer Concerns, and How I Use It
Cardarine is one of my favorite research compounds, and I haven't talked about it in a while. I cycle on and off of it for reasons I'll get into. It does great things for endurance, lipids, and metabolic health. But there's also that cancer conversation we have to address head-on.
What Cardarine Actually Is
Cardarine, also known as GW501516 or sometimes Endurabol, is a PPAR Delta agonist. It's not a SARM, even though a lot of SARMs websites sell it. It doesn't touch androgen receptors. It doesn't affect testosterone or estrogen.
GlaxoSmithKline and Ligand Pharmaceuticals developed it in the 90s as an "exercise in a pill." It went through phase one and phase two trials for hyperlipidemia and metabolic syndrome.
Then in 2007, GSK abandoned it after rodent studies showed cancer onset. WADA banned it in 2009. Today it lives in the research chemical and gray market world.
How It Works
Cardarine binds PPAR Delta with very high selectivity, over a thousand fold compared to PPAR alpha and gamma.
In skeletal muscle, this activation recruits PGC-1 alpha and upregulates fatty acid transport, beta oxidation, and mitochondrial respiration. Your muscle fibers start preferentially burning fat instead of glucose. They take on characteristics of fatigue-resistant type one fibers.
In the liver, it promotes fatty acid uptake and raises HDL while clearing VLDL and LDL particles.
In adipose tissue, it shifts the balance toward burning fat over storing it.
There's also an anti-inflammatory effect in macrophages and the vasculature.
What the Human Studies Show
A 12-week placebo-controlled trial in 268 subjects with low HDL tested doses of 2.5, 5, and 10 mg daily. At 10 mg, HDL went up 16%. LDL dropped 7%. Triglycerides fell almost 17%. ApoB dropped almost 15%. Free fatty acids dropped around 19%.
The lipoprotein analysis also showed a shift away from small dense LDL toward larger buoyant LDL particles, which is a real cardiovascular win.
A separate two-week trial in moderately obese men using 10 mg daily produced massive changes in just 14 days. Triglycerides down 30%. LDL down 23%. ApoB down 26%. Fasting insulin down 11%. Liver fat content down 20%.
That liver fat number is wild for two weeks.
What You Actually Feel
The endurance effect is the hallmark. When I take Cardarine and do cardio about 30 minutes later, my cardio is so easy I barely think about it.
Within a week or two, cardio feels easier. Distances that used to be exhausting become manageable.
You also get a gradual decrease in body fat, especially when paired with training and a real diet. It's not like a GLP-1 or true tirzepatide. But it slots into a fat loss stack nicely because it's working through a different pathway than most of the other tools.
There's no stimulant feeling. No jitters. No crash when you stop. Just steady, easier energy.
The Cancer Conversation
Here's the part everyone wants to know about.
A two-year rodent study found that all tested doses caused cancers in the liver, bladder, stomach, skin, thyroid, tongue, testes, ovaries, and uterus. The tumors came on faster than typical carcinogenicity assays show.
The doses in those rodents roughly correspond to 20 mg per day in a very small human, maybe 100 pounds. Scaled to a 220-pound person, you're looking closer to 50 to 75 mg per day.
I'm not going to tell you it does cause cancer. I'm not going to tell you it doesn't. We have conflicting data. There are also studies showing anti-cancer effects of PPAR Delta activation. We don't have human cancer data.
Am I a little suspicious that something which beats statins on lipid improvements got shelved? Sure. I notice patterns. I'm not making accusations, just thinking out loud.
What I do is respect the dose and cycle it.
How I Use It
For me, the safe daily dose is 10 mg. You could go to 20, but I don't feel any outsized benefit from doubling up.
I run an 8-week cycle, then take 8 to 12 weeks off. I'm not relying on Cardarine alone. I'm pairing it with things like MOTS-c and SLU-PP-332, which work through related but distinct pathways. I'd feel safer running 10 mg of Cardarine alongside MOTS-c than running 20 mg of Cardarine by itself.
For longevity dosing, 2.5 to 5 mg daily is plenty. You still get lipid improvements without pushing the dose into questionable territory.
The half-life is 16 to 24 hours, so once-daily oral dosing works fine.
If you compete in a tested sport, do not touch this. It has a long detection window and WADA put out explicit warnings.
A Quick Note on GW0742
There's a related compound called GW0742 that's basically an enhanced version of Cardarine. Milligram for milligram, it feels a bit stronger to me, especially on the cardio side. Less human data on it though. Let me know in the comments if you want a full video on GW0742.
My take
I love Cardarine. The endurance effect is real and noticeable within a couple weeks. The lipid improvements rival what you see with statins. Used at 10 mg in 8-week cycles, with time off between, it fits a spot in a fat loss or longevity stack that almost nothing else fills. The cancer data is real, but it's also conflicting, and the rodent doses scaled high. I'm not telling you to use it or avoid it. I am telling you that if you do use it, respect the dose, cycle it, and don't rely on it as your only tool.
Full transcript click any paragraph to jump video
Hey everybody, this is Hunter Williams. I hope you're doing amazing wherever you might be in the world. Today's video is going to be about one of my favorite research compounds that I haven't talked about in a long time and I don't really talk about, but I love it. And I use it pretty frequently, I would say cycling on, cycling off for reasons that'll talk. But that compound is Carterine. It is also known as GW501516. But Carterine is a PPAR agonist that does a lot of amazing things, namely it enhances endurance.
So it's a favorite of endurance athletes or athletes who just are looking to improve performance, specifically cardio performance. probably mild strength gains, but more so cardio performance. And then also too, there's pretty good data around what it does for our lipid profiles. So when it comes to improving lipids profiles, raising HDL, lowering LDL. There's a lot of good around that as well. It was originally a product, I think of GlaxoSmithClimb, which is actually not too far down the road from me where their headquarters is,
But it was a part from them. They discontinued it. I'm going to talk a little bit about that today. If you're familiar with carterine, you are probably familiar, with some of the, let's say hesitation around it because of, the studies that they did that it supposedly caused cancer in rodents. We don't have any human data on whether or not it causes cancer. we do have a lot of anecdotal data to show that, it probably is not causing cancer however, I want to not throw it off and we'll talk a little bit about that today. Spoiler, I'm not going to tell you one way or the other. If it does or doesn't cause cancer, and then talk about it and you can kind of go from there and make an informed decision.
But I personally use Carterine in a cyclical manner. I love it. It does a lot of great things for my health, but also for performance. perspective, and that's why I like it so much. Pretty affordable too, pretty easy to source in the research world. But anyway, that what we're going to talk about today. As always, before I jump in, make sure you're on the email list. You can sign up for the peptide cheat sheet or just join the e-mail list, the link will be down in description below. And you can also Check out now my private group is called the Axion Collective. We've got over 200 members in there. So it's rocking and rolling there you can join and cancel any time, but I really think you'd like it.
You can direct message me, you could talk with all the other members and the forum and we do live calls. If you want to make sure that your questions are answered, come on with your camera on or off and talk to me on Thursday nights at 8 p.m. Eastern. Without further ado, I'm gonna share my screen and today we are going to dive into Cardiff. All right, let's get into it. We're going to do a comprehensive overview of Carterine today. Again, it's called GW 501 516, but basically it is a synthetic PPAR Gamma agonist, initially developed as a drug for metabolic and cardiovascular disease.
And first, as always, I like to give you a little bit of background and intro. So Carterin or GW501-516. I've also in the research, some people call it Endurable. It's not an anabolic steroid, Typically you see a lot of anabolic steroids with the ending, the BOL in the end. But anyways, some people out there have called it Enduroval. It's an investigational drug originally developed in 90s through a collab between Glaxo and Ligand of pharmaceuticals. The goal is to create exercise in a pill to which back then was kind of a novel idea.
We have a lotta more things now to do really good with that. including MOTC and SLU and some of the mitochondrial stimulants, but I still think carderine has a place in those because it works a little bit differently than those. But again, it was a compounded treatment of block diseases such as obesity, type two diabetes, dyslipidemia and cardiovascular conditions. So we saw it developed in the 90s, mid 2000s phase one and phase two clinical trials for hyperlipidemia and metabolic syndrome. 2007, Glaxo abandons development after animal studies reveal rapid cancer onset.
2009, WADA adds carterine to the banned substance list. 2013, water issues explicit health warnings and cancer risk. So if you are an athlete, you better not use cartering. Despite the abandonment, it gained notoriety as a black market, gray market performance enhancing drug after studies suggested it could dramatically boost endurance and fat burning. Today it is illegal for athletic use, at least by sport organizations and sold as research chemical on the gray markets, but there are still people attracted to it and I even would dare say potentially could use it in a longevity protocol.
Now let's look at some of the mechanism of action. Again, we have this, excuse me, I called it PPR Gamma earlier. It's actually PPAR Delta agonist, but sorry, sometimes I get the Latin letters confused, or the Greek, the greek or Latin. I'm making a fool of myself when it comes to history right now. But anyway, carterine is a high potent selective agonist of the PPAR Delta, which is nuclear hormone receptor regulating gene expression in various tissues
binds the PPR Delta with sub-nanomolar affinity and activates with greater than a thousand fold selectivity over the ppr alpha and ppar gamma isoforms. By activating PPR Delta, carterine induces transcription of numerous genes involved in fatty acid metabolism, energy expenditure, and cellular differentiation. Now what that means is that we have muscle reprogramming, so in skeletal muscle, PPAR Delta activation recruits the co-activator PGC1 alpha which again
thank metabolic enhancer and exercise mimetic that also is going on with SLU up regulating proteins for fatty acid transport beta oxidation and mitochondrial respiration muscle fibers preferentially burn fat for energy instead of glucose taking on the characteristics of a fatigue resistant type one fibers. We also have systemic effects. So beyond muscle PPR Delta is expressed in the liver adipose tissue and other organs and liver activation promotes uptake and utilization of fatty acids
and increases proteins that raise HDL cholesterol and enhance the clearance of VLDL and LDL particles, which we would normally associate as a bad cholesterol. I know that's probably the wrong phrase for those, but anyway, just for brevity, the sake of brevety. And then there's no hormonal impact. So unlike anabolic agents, carterine does not act on androgen receptors or hormones. and its performance effects come from metabolic modulation rather than muscle hypertrophy. And again, when we look at sex specific, this is one that the dosing and everything for men and women is going to really be the same because there is no
sex-specific dosage or effect on androgen receptors or anything like that. And beyond muscle, PPR Delta is expressed in the liver adipose tissue in other organs. So, Carterine's activation of this receptor has systemic metabolic effects. We have liver effects, PPAR Delta influences lipid homeostasis, and the activation PDR Delta through Carterin promotes uptake and utilization of fatty acids and increases the expression of proteins that raise HDL cholesterol and enhance, again, VLDL. LDL clearance. We also have adipose tissue PPR Delta activation increases genes for fatty acid utilization and may shift the balance toward fat burning over storage,
meaning that we will burn those fatty acids instead of storing them. And then there is an anti-inflammatory effect PDR Delta has anti inflammatory roles in macrophages and our vasculature suggesting potential benefit in reducing heart disease or atherosclerosis. The mechanism can be summarized as enhanced fatty acid oxidation and energy expenditure in muscle and other tissues, which leads to an improved metabolic profile overall. Now, practically speaking, when we're working on all of those pathways, as mentioned, what does that do for us?
When we have enhanced endurance, there was a landmark study that showed that PPAR or Delta activation via Cartering can mimic some of the effects of exercise mice given carterine for several weeks were able to run nearly twice the running distance of control mice without prior training, and the drug promoted a shift towards slow-twitch muscle fibers and increased oxidative enzymes. So if you're a sprinter or someone that relies on power, maybe not the best thing because it does have a preference for those slow, slow twitch muscle When we look at fat loss and metabolic syndrome, it showed therapeutic effects and rodent models of obesity and insulin resistance in high-fat,
diet-fed mice, PPR on delta activation, increased metabolic rate, fatty acid beta oxidation, and mitochondrial proliferation while reducing fat or adiposity. And treated mice had lower fat accumulation in muscle and gained less weight despite similar food intake. We also see improved insulin sensitivity, carderine ameliorated, diet induced insulin resistance, treated obese mice had significantly lower fasting glucose and insulin levels and improved glucose tolerance test. Even in genetically diabetic mice, carterine lowered blood glucose, insulin, and then lipid profile improvements in diet-induced obese rhesus monkeys.
Carterin increased HDL cholesterol and decreased triglycerides and fasting insulin without actually affecting blood glucose. Which is pretty cool. Now we move to the human studies, which there are very limited of, however relevant nonetheless. So there was phase one and phase two trials. There was a lipid and cholesterol effect trial. It was 12 week trial, large placebo controlled trial in 268 subjects with low HDL cholesterol examined, carterina doses of 2.5, 5 or 10 milligrams daily for 12 weeks.
and at the highest dose at 10 milligrams daily, HDL increased by 16%, pretty powerful. LDL cholesterol decreased by 7%, not as powerful, but still relevant. Triglycerides, which is very important when we look at inflammation, inflammatory fat, visceral fat decreased 16.9%. APO-B, I think much more relevant to the cholesterol conversation, decreased 14.90% and free fatty acids decreased around 19%. The lipoprotein subclass analysis revealed a shift towards less atherogenic particles, BLDL and LDL were reduced, small dense LDl particles reduced and total HDL particles increased.
Then there was a metabolic effects in obese volunteers trials, a two week trial again, which you know, it's like two weeks. Is that really enough to test some of this stuff? There was obviously changes in the parameters, but I would love to see this played over like, you know, at least 12 to 16 weeks. Randomized trial and moderately obese, otherwise healthy men tested Carterian 10 milligrams a day for two weeks, despite the short duration produced significant metabolic improvements. Fasting triglycerides dropped by 30%. LDL cholesterol decreased 23% in this case in just two ApoB decreased 26%, fasting insulin reduced 11%, 20% reduction
in liver fat content, which I think is huge to see a 20 percent reduction and the amount of liver that someone has in their body. In some cases, I mean, that's crazy how much fat is coming off in a short amount And then there was reduced oxidative stress, so urinary isoprostanes fell by around 30%, which just indicated reduced oxygen stress on the body. And a muscle biopsy confirmed upregulation of CPT1B, which is a key enzyme in fat transport into the mitochondria, and Carterine reversed multiple abnormalities
associated with metabolic syndrome in these obese men. Pretty cool there. And again, I don't know specifically how much these people were exercising, but those would probably be on par with changes that pretty vigorous exercise would do, or inducing it with Carterine. When we look at aggregate, what does that look like? So when we sum all those up, key outcomes, 70% on average HDL increase, which is our good cholesterol, 30% triglyceride reduction on an average,
20% liver fat decreased, 11% insulin improvement, modest reductions in LDL we're seeing, and more importantly, a shift away from small dense LDl towards larger buoyant LD L was documented. Small dense LL is more atherogenic, meaning it's more likely to cause heart disease, thus this remodeling. It implies a cardiovascular risk reduction. And then animal studies showed protection against diet fatty liver and reduction of liver inflammation markers, lower isoprostanes and possibly lower CRP aligned with the idea that it may quell inflammatory pathways associated with obesity.
Now, would, with carterine be my first thing to reduce your CRB and lower inflammation? No, but we're going to get some benefits along the way of using it. I think we use it intelligently. There definitely is a reason and a use case for it, But basically, in the human studies, we saw better lipid profiles, enhanced fat utilization, and overall signs of improved metabolic health. But we do have to look at some of the safety concerns. So in big red letters, does carterine cause cancer? Well, let's look into what was found originally, because there's actually some studies that show that cartering was anti-cancer.
However, there was this cancer. Now, of course, I'm kind of like a conspiracy guy. I think if you operate in the world that we operate and you kind have just noticed patterns. And I wonder, okay, if we're looking at this, what seems to have better lipid profile improvements than statin medications, which is a multi billion dollar industry, do we want something to necessarily threaten that multi-billion dollar Probably not. Also, if you're a pharmaceutical industry too, which I don't know if Glaxo and Ligand produce statins, but would you want to make a product that would
be cheaper than the statens and also cannibalize the sales? So I'm not saying anything particular, it's just something I like to think about. Nonetheless, let's look at this cancer study. A two-year rodent study revealed that all tested doses rats develop cancers in the liver, bladder, stomach, skin, thyroid, tongue, testes, ovaries, and womb. These tumors arose faster than typically seen in standard carcinogenicity assays, suggesting a potent cancer protein effect. And the doses in rodents correspond roughly to a 20 milligram a day dose in humans. In some cases, It really, that would be a very light human.
So that's probably closer to like a 100 pound human using 20 milligrams. I think if you go up to a 220 pound, human, it's closer. To somewhere in the neighborhood of like 50 to 75 milligrams a day. That's where we get this. It's not safe, but again, there's lots of studies out there that show that there were. anti-cancer effects. Now, what do we do with that? I think we just use it for what it is. For me, the safe dose per day is 10 milligrams. Sure, you could go to 20 milligrams, but I really keep it, I like to keep in 10mg in that range because one, it's a benefit to it.
Two, don't feel any outsized benefit of going up to twenty milligrams versus 10 mg. And three, this isn't something that I'm solely relying on because I am using other things. You know, we have all these other things out there now that maybe or maybe not, they cause cancer, but at least there's no documented evidence in some of these things like MOTC or SLU. So I'm not saying they do or they don't, But I would feel a little bit safer if I used Motsi with 10 milligrams a day of Carterine as opposed to 20 milligrams Carterin without any MotC. But basically this findings led GSK and regulators to conclude that long-term use in humans could be carcinogenic, keyword could.
As a result, all clinical trials were terminated and developed and ceased once the cancer risk became evident. But again, we don't see that in. practice with humans. Short term trials, carterine was well tolerated with no significant adverse events relative to placebo doses up to 10 milligrams for two to 12 weeks. Trials did not note major liver enzyme elevations, no changes in blood pressure and no clinical symptoms. While liver enzymes were not primarily elevated in human trials. Cost remains at longer exposure could potentially lead to liver issues because PPR Delta agonists Activation had some patients develop liver enzyme spikes
after longer treatment. And then animal reproductive studies indicated fetal toxicity and birth defects at super high doses. High doses of Cartering caused increased embryo death and developed abnormalities in rats. Again, not seen in humans, but definitely wouldn't want to be used in a pregnant person. Again, short-term use of cardinine did not reveal immediate toxicities in humans, but there is that long- term cancer risk. And again, when we look at the cancer thing, there's evidence in both directions. So I just want people to be aware. I'm not saying it doesn't, But I am not say it does. We just have to aware, and again you can get cancer from living near a nuclear plant.
There's like so many ways you could get a cancer that it's hard to control some of these variables for. But it is out there via the mechanism we did see it in rodent studies. Now looking at dosage and usage patterns in formal research, it was administered orally once daily, and it is very, at least as far as we know, available orably. 2.5 milligrams to 10 milligrams per day, the 10 milligram dose was generally the highest used in phase two trials. The half-life is reported to be around 16 to 24 hours, which allows for once-daily dosing.
Therefore, five to two milligrams can be considered a clinical dose range that was explored for Potential therapy. Now among athletes, bodybuilders, self-experimenters people usually will start at 10 milligrams and go up to 20 milligrams. Again, I think 10 milligram is plenty. The most commonly sized regimen is 10 mg twice daily or simply 20 mg once daily. And I to the cancer conversation, is this something that I would use all the time? No, no. I use this in an eight week cycle. Every day for eight weeks and then just take another eight to 12 weeks off.
So we see clinical range, two and a half to 10 milligrams, athletic use 10 to 20 milligrams. And then for longevity protocols, I think for those lipid panel improvements, maybe they're not as drastic as they would be at 10mg. But I, think if you're doing two to five milligrams a day, so two-and-a-half to 5mgs a, day that would, be kind of my microdosing improvement in long-term health without really any sort of risk that I would see popping up. But Carterine is orally bioavailable and was given as a pill in trials. It's quite stable and has a long detection window in the body.
So again, if you are getting tested for your sport, and that's an issue, I would probably stay away from it. And I think there's other things that you could use that I probably wouldn't talk about publicly because I'm not in a business of helping people get around drug tests for their sport. What can we expect? One, we're going to see improved endurance and stamina. This is the hallmarked effect. So this is what you really notice. this Is why I like taking it is because when I say Carterine and then do cardio, you know, 30 minutes after taking Carterin, my cardio is so easy. Like I just don't even think about it.
That's how good it Is. so within a week or two cardio feels easier, distances or times that used to be exhausting become manageable, obviously increased fat burning. I won't say that this anywhere close to a GLP, like we're at a true tide. However, it can definitely help in those. And I think in the process of building a fat loss stack would be really good. So users often report a gradual decrease in body fat percentage, especially combined with exercise and proper diet. Many describe it as accelerating fat lost, but not in a stimulant way. I don't think that's the cool thing too, is it's not a stimulus.
Better lipid profile, obviously we went over that. No hormonal effects, so it's not going to affect testosterone, estrogen. Oftentimes it is lumped in as a SARM, which is a selective androgen receptor modulator. It is not a SAM. So if you see it sold as the SAR, it it not as SAR. A lot of SARMs websites sell it, but it s not SARm. Subjective of all being obviously during this time, energy is usually better You feel fitter during the day. There's no nervous system simulation. And so you get a little bit more energy, but you obviously it's just much more of a kind of feeling good, which are not stimulated.
Again, lack of acute side effects in the short run. Most recreational users do not experience adverse symptoms. I think to the cancer point, it just like cycle on and cycle off and you probably will be okay. After discontinuation, users generally do not report a crash as you might with hormones or stimulants and endurance capabilities likely return to baseline over weeks as muscle gene expression normalizes. But I think the biggest thing is like if you take it for a few weeks and your cardio is really easy, it doesn't mean that your cardio goes back to being
really hard if it was hard before, but it is not going to be as easy as it. Was before. So there's no crash, which you also will probably notice like, Hey, maybe it's a little bit harder for me to get through my cardio than it was before. But again, you do, if you're a pro athlete or in any sort of organization that's tested, do you have to worry about it? And again it is a research chemical, so just be aware of that. Make sure you know where you are getting it from. Carterine is fascinating molecule. When we look at the side of the promise, we obviously have the metabolic benefits, improve cholesterol and increase your endurance. I would say like if those two things are relevant to you, it absolutely conversation to have.
improving cholesterol, you know, without having to use a statin or anything like that, and then increasing endurance. But we do have that cancer risk, that could have come along with it. Obviously, I think from a longevity perspective, when we look at those lipid profile improvements, we looked at what's going on there, a low dose is absolutely fine in the context of longevity. And just if someone has some higher cholesterol markers, especially in LDL, APOB side of things, it could be really good. Showed great promise, but the cancer risk kind of killed it, at least from a pharmaceutical perspective.
Who knows how, I guess, true that cancer was or not. We'll never really know. It won't be something that's brought to market pharmaceutically. But in the research world, it will probably continue to be used amongst other things, and I think cycled in with some other peptides like SLU, MotC, things of that nature. And that is it for the slides. And, that, is my overview of Carterine. So, it's been a while since I talked about Carterin. Now, there is another compound called GW0742. I've actually been thinking about making a video on that one because that's kind of like an enhanced version of Carterine,
so if people like this video, I can definitely do that and I'd love to hear your feedback in the comments if you want me to talk about GW0742, There's much less human data around GW 0742. I think milligram for milligram, it's probably a little bit stronger than carterine. And notice a bit more in my cardio in terms of the benefit from it. My lipid panels are pretty good as far as I know. So I don't notice anything there that would, um, necessarily be shifted from it outsized to Carterine. But I would just say from a performance enhancement standpoint, the GW0742 is better.
I love Carterin. Again, I don't like to tell people one way or the other, hey, this causes cancer, stay away from that. Or no, it doesn't cause cancer. You don' have to worry about that because we have conflicting data on this. So there's there's studies that show that it actually could be anti-cancer when there studies show it could. Again, I think the dosing is probably relevant too. I definitely wouldn't use 50 milligrams a day of Carterine, or if I did, it would be for a very, very short window, but I love Carterin. It has a good place. And we look at building out a comprehensive fat loss stack, which I didn't really go into today.
When we looked out building that stack Carterins is kind of this one that fits in a place that not so many of the other peptides or hormones or whatever we're doing exist within, which is why I think it's relevant because we can add it in and get this added benefit that's not going to overlap with some of the other peptides that we know love and work really well for fat loss. But thank you guys so much. Just in closing, I always like to say you are the reason I exist. So without your support, don't get to do this. Thank you so, much and whether it is using my code, being on the email list, in the groups, whatever it, is help sharing this with your friends and family.
That goes so far in helping me to be able to bring these messages to you, which hopefully are helpful and improve your life. That's my mission with this. So I get fired up to get out of bed every morning to bringing these message to and I wouldn't be to do that without you. Thank you from the bottom of my heart. I have so much gratitude for you guys out there that help support this and ultimately will help this because this is not about me, it's not me as an educator or influencer or whatever I am. It's about helping people improve their lives for the better, improve health and make a difference in the world and help lift other jobs.
And that's it for this one today. I will see you in the next one. Peace.
