ATX-304 | The AMPK Activator That Mimics Exercise

0:10 Hey everybody, this is Hunter Williams. I hope you're doing amazing wherever you are at in the world. Today's video is going to be about a small molecule compound known as ATX304. And I guess I'll go ahead and clear up the confusion because you may see this sold as a compound that is OS01 or even in some cases OS03. So I believe what happened, and this was just based on what I've heard. I don't know if this is the 100% accurate story, but this compound was actually originally developed as ATX 304. And I think someone else also developed

0:46 it as OS 01. and there's a skincare company that is called OS01. To be honest, I Don't Know if the skincare Company uses this molecule in it. But anyway, the people that sell it either is ATx or OS-01 had some sort of trademark on it. So you're going to see it as both names out there, but just know that we're talking about the same compound. For the purposes of this video, I'm just going call it ATX304, so as not to confuse it with the skincare product OS01, which again, it might, i don't know if that contains this compound that were talking, about but either way we are talking a capsule product that is basically an AMPK activator.

1:23 A little bit of background on it. We came across this a few months ago, tested it in ourselves, got really good results, and now we sell it as a product at BioLongevity Labs called BioAMP. And we also have an enhanced version called bioAMPMax that has 5-amino-1-MQ and a couple other different ingredients in it to just help the absorption and help with energy and fat loss. But I really love this product. And to be honest, it's almost to the point of getting a little spoiled in the sense of what we have access to now. Yes, I understand a lot of this stuff is expensive. In our case, we're one of the first companies to bring this to market.

1:54 So when you look at the supply chain, It's really expensive to get these products out into the hands of people. Now, my hope is that as companies like us bring the market, more companies come into it because there's competition and obviously more demand. That drives the price down. That is my goal in doing all of this. Believe me, if it were profitable or it was sustainable, I would give these things away because they are that much of a game changer for human health. Now I know that sounds rich coming from someone that is affiliated with companies that sell this, But I just want you to understand when you're the first person to bring something in the market, it's expensive to do so because there's no supply chain built and there is no demand for it.

2:28 And so hopefully as this gets more and more out there, more people will demand this and the price comes down. That is my goal in doing this. So just know that that's kind of how capitalism and free markets work. But that being said, ATX is a really really game changer project product and again we're kind of spoiled because we have all these crazy products coming out that work really well and that's what you know the next couple months my videos are going to be about is i i barely have time that day to do the research and the videos on things that we Have coming, out so that is what my next focus is going be in the coming months but anyway to atx it's really amazing

3:01 it is an ampk activator so it works slightly different than some of these mitochondrial stimulant supplements the mitochondria Energy will be a byproduct of it, but it's a little bit different. So that's what we're going to go into today. I'm going cover everything about it. Cover the mechanisms, the mouse trials, a few bit of human data that we have on this is also as well as the dosage that I would recommend based on like what I've experienced and then also to what That's what we're going to talk about today. Hopefully it will be informative and educational at the same time and also maybe a little entertaining.

3:35 But don't forget the peptide cheat sheet is down in the description below. You can download that at any time. If you want access to all these amazing things, the best place to be is on the email list because I send out a few emails per week to keep you updated. with sales and all the things out there on these products. And also too, if you want to be in the best private community on the planet, check out Fully Optimized Health down in description below and that's $99 a month. You can cancel anytime. So without further ado, I'm gonna show my screen and today we're gonna learn about ATX 304. All right, my name is Hunter Williams and today we're going to be covering ATX 304. As you can see, we sell this as Bioamp.

4:07 There's other companies that sell it. You should check it out. But I really, really like this product and it basically is going be a deep dive into this AMPK activator. So just a little background on this. It was originally discovered at Umea and hopefully I'm pronouncing that right. University in Sweden. and bet a gene on AB through screens for compounds that increase AMPK activity. So early preclinical studies showed it could potently improve metabolic parameters in obese and diabetic mice. A single center phase 2A trials tested 0304, same thing, ATX304030.

4:42 And 65 patients with type 2 diabetes on metformin, establishing proof of concept for its glucose, lowering, and vascular effects. So the 28 day trial, and we'll talk a little bit about that more, showed significant reductions in fasting plasma glucose and insulin resistance with no safety signals or side effects observed, which is pretty cool, especially relative to some of the GLP-1s and other things we have out there. And so following the promising results, Cambrian Biotech acquired the intellectual property for it in 2023 and launched amplifier therapeutics to advanced 0304,

5:16 which then was renamed ATX 304. Which is what we know now for today for cardio metabolic and aging related diseases. So originally tested for diabetes. Now we see the anti-aging effects. As of 2025 today, It has completed phase one studies in obesity and entered phase two development, making it one of the first direct AMPK activators to reach clinical stage trials of metabolic disease. And thankfully for us, all of these crazy cowboy researchers out there right now are testing it and putting it to good use for fat loss.

5:46 So let's look at what AMP does. Why would we want to activate AMP in the 1st place? As you probably know by now, I like to break things down as simple as possible. And when we look at AMPK, I know because before I learned all this stuff or taught myself all the stuff, you know, was into it. I would hear people on podcasts say AMP, AMP. Oh, that sounds cool. Right. Well, let's understand what it does at like a third grade level. So AMP K stands for AMP Activated Protein Kinase.

6:16 It functions as the body's master energy regulator. detects cellular energy status and triggers adaptive responses when energy is low, such as during exercise or fasting. So when we activate AMPK, what does it do? It increases glucose uptake in the muscles to help us build muscle. It enhances fat burning through fatty acid oxidation. it reduces fat and cholesterol synthesis, bringing down our cholesterol, it improves insulin sensitivity, and it also promotes cellular cleanup processes, just known as autophagy, which everyone loves, right?

6:48 So these actions collectively shift the body from energy storage to energy utilization. Why are people fat? Because they're storing too much energy and not using the energy from all the food that they are eating and amongst other things. So it's similar to what happens during exercise. When we talk about AMTK, we're really talking about inducing a state of exercise in the otherwise known as an exercise mimetic. So let's look at the mechanism of action. With ATX304, this is what makes it unique.

7:18 It prevents dephosphorylation. Now, what does that mean? Unlike classical AMPK agonists that mimic AMP, ATx305 increases AMP activity by preventing de-phosferization of the critical threonine 172 residue on the AMPk alpha subunit. So what that does is it maintains active state. So it suppresses protein phosphatase 2C, PP2C mediated removal of PTHR 172, thereby sustaining a AMPK in the phosphorated active-state.

7:51 And what this does, is a preserves energy balance. This activation does not require a drop in cellular ATP. It boosts PAMPK without causing an energy deficit in cells. So when we sum all that up, ATX locks AMPK in its on position, triggering downstream energy stress responses as if the cell were exercising or calorically restricted. So again, all of those are really confusing chemical terms. You don't need to understand all them. Just know that ATx takes this AMPk and says, okay, we're going to turn this on now and it's going make the body basically go into an exercise state or

8:28 a calorically restricted state into which we are burning more energy. So let's look at this AMPK activation and mitochondrial activation, because like I said in the intro, there is a little bit of mitochondria activation as well. So beyond direct kinase activation ATX also acts as a mild mitochondrio activator. It induces modest mitochondrion coupling, not a ton, but modest. it raises basal oxygen consumption by around 38% and it increases cellular respiration rates.

9:00 What does that mean for us? It increases energy expenditure, obviously increasing metabolism, it raises AMP and ADP levels, and it further engages AMPK's energy sensing pathway. So for this reason, ATX is described as a dual AMP K and mitochondrial activator, essentially mimicking exercise at the cellular level. Remember I said it's an AMP-K activators, but as result of that AMP it is actually improving mitochondrial function and mitochondria health and yes we have the mild mitochondrion uncoupling but long term that's going to help us out so let's look at some of the downstream effects on glucose metabolism

9:34 so it promotes insulin independent glucose uptake into skeletal muscle and vitro it increased to the oxyglucose uptick in muscle cells in an AMPK dependent manner It also improves insulin sensitivity like I talked about. So by clearing blood glucose into muscle, it reduces the demand on pancreatic beta cells and in diabetic mice, ATX lowered hyperglycemia and improved insulin resistance and beta cell stress. And also treated animals and humans show reduced fasting glucose and insulin levels leading to improved HOMA-IR, which is an insulin-resistance index.

10:08 Also notice there, not only did it improve fasting in glucose, but it also improved fasting insulin level. A lot of things can maybe improve fasting glucose transiently, but they won't over time necessarily improve our fasting insulin, which to me is a better indicator of metabolic health than just the fasting-glucose alone. What that means is that the insulin sensitizing effect of ATX basically is a hallmark of AMPK activation as muscle and other tissues become more efficient at glucose utilization. And again, even if we're exercising, if ever we are dieting right over time, we become less insulin sensitive because of aging.

10:42 Now we gonna look at some of those anti-aging effects that are gonna come as a result of this. So let's look lipid metabolism. We have increased fat burning. When we activate AMP it switches cells from anabolic to catabolic mode. Now that might sound bad at first, but hear me out. ATX3304 causes the phosphorylation of acetyl-CoA carboxylase, thus relieving its block on fatty acid oxidation. So in liver and muscle, ATx303 increases fatty-acid beta oxidation and concurrently suppresses lipogenesis.

11:12 A recent study in a fatty liver disease model showed that it shifted hepatic programming of the liver toward fat burning and gene expression of lipid oxidation pathways rose while lipids synthesis pathways were downregulated. and treated mice had markedly lower liver triglyceride content and diminished fatty liver compared to control. So again, all those things to say we're improving liver health by doing so. Now let's look at this effect on cholesterol. ATX 304 normalized blood cholesterol in diet-induced hyperlipidemic mice, which just means high cholesterol, reducing cholesterol for around 212 milligrams

11:47 per deciliter, Which in my opinion for humans is not Something you have to worry about from your total cholesterol, but it brought it down from 212 to 143. And again, when we have the statin conversation, why would we not use something like this as opposed to a stat? Maybe cost is an issue because statins are given away because they kill you faster. But we can use this to get better effect on our cholesterol without the downside of statens as we're going to see in the safety profile for what we know about this so far. We have a 45% cholesterol reduction. We have a reduction of fat mass.

12:17 So visibly ATX fed mice exhibited reduced adiposity. Total body fat mask drops significantly approaching the lean control levels even when food intake was not continuously reduced. It was on the same amount of food that they ended up being almost as lean as the controls of the mice that were not induced to be fat. We also have muscle preservation. I think this is where a lot of these small molecules that we're gonna see are actually going to compliment or even outshine GLPs is in the muscle presentation.

12:49 It's almost impossible to lose fat without losing muscle. What we want to see though is the proportion of fat that we, or, you know, weight that lose, be as much fat as possible and as little muscle as possibly. Because the thing about this, if you lose a hundred pounds and 50% of that is fat and 15% is muscle, are you really that much better off than you were before? You have the same body composition. You've lost weight, but if your 50 percent fat, and fifty percent muscle. Are you still not 50% fat? So yes, is it better to be a lower body weight?

13:20 Yeah, but if we don't shift the composition, we're not getting the actual intended effect for our health. And so weight loss under ATX 304 comes without muscle loss. So studies note preferential fat reduction and even potential muscle gain or improved exercise capacity. Again, when we look at the pathways, think about the pathway. GLP-1 pathway, which is arguably a great thing to do. I'm not saying against that versus AMPK activation. You're getting the intended result, fat loss without the consequence of muscle loss, it's crazy.

13:55 And I think we're going to see a shift in the next five years of improving upon the GLPs ones to get this. basically fat loss without the muscle loss and potentially even muscle gain. So it flips the metabolic switch to a fat-burning high energy state akin to vigorous exercise or caloric restriction, which is pretty cool. Now, let's look at the cellular stress response. Again, we're inducing exercise in the body, right, or a state of exercise and body. AMPK activation induces pro-survival pathways like autophagy, so in beta cells, ATX enhanced autrophagic flux and protected cells from toxic protein aggregates.

14:28 In a mouse model over-expressing human islet amyloid polypeptide, chronic ATx304 dramatically reduced pancreatic islets amyloid deposits which are linked to beta cell failure in type 2 diabetes. It also prevented induction of unfolded protein response genes in islets under high glucose stress. And in the fatty liver model, it markedly reduced markers of oxidative stress and lipid peroxidation in liver. So what this suggests is that ATX3 or 4 can relieve endoplasmic and oxidated stress in metabolic tissues, which is pretty cool.

14:59 Now let's look at some of these vascular and cardiac effects. Again, because what do we have is like the four horsemen, right? Cardiac disease, number one. We have improved microvascular perfusion. And so when we look at like diabetes and neuropathy, what is the consequence downstream from that? Vascular impairment. So in both animal models and diabetic patients, treatment led to significantly improved capillary blood flow, which is only gonna enhance everything. We have enhanced cardiac function, so in obese mice, ATX activated cardiac AMPK, increasing myocardial glucose uptake and reducing pathological glycogen

15:34 buildup in heart tissue. You also had exercise-like adaptations. So treated mice showed improved left ventricular stroke volume and increased end-diastolic volume with a corresponding reduction in resting heart rate, changes characteristic of an endurance exercise adaptation without the exercise. And notably, and I think this is really important, for what we know so far, it did not cause any increase in heart weight, not rate, but weight with a W, which basically means that it not caused heart enlargement like a lot of times over exercise cam and mice or rats and unlike

16:05 some prior MPK activators that induced undesirable cardiac enlargement. So by boosting cardiac energy efficiency and peripheral circulation, It supports cardiovascular function akin to an exercise mimetic. Now let's look at some of the studies around diabetes. So in diet induced obese mice. ATX improved metabolic outcomes so treated obese mice had lower blood glucose and insulin levels. So obese my switch to feed containing ATx were prevented from progressing to overt diabetes despite continued high fat diet feeding.

16:36 They had lowered fasting glucose, insulin and Insulin resistant index even without inducing weight loss in the short term and over longer term treatment moderate weight and fat mass reductions occurred alongside metabolic benefits. It also improved cardiovascular measures like we just talked about. So a significantly increased exercise endurance and cardiac output while reducing heart rate, essentially reproducing the effects of exercise training. Pretty cool there to see the reduction in heart rates, because a lot of times when we talk about fat loss, we're at a true tide.

17:06 What we know happens, increased heartrate because of the central nervous system activation and a host of other things going on. Let's also look at aging mice. So they took middle-aged mice and they fed ATX chow for up to 12 months starting at six months old and then ATx prevented age-related metabolic decline so treated mice did not develop the rising insulin levels and insulin resistance that occurred in controlling or the control age mice Not only this, but it reversed established hyperinsulinemia in older mice indicating improved systemic insulin sensitivity.

17:41 And then age mice on ATX showed enhanced exercise capacity on treadmill tests and better cardiac function compared to untreated controls. So this indicates that it can mitigate multiple aspects of aging, which again, if you look at aging if, you're looking at inflammation aging. What do we have? We have metabolic dysfunction, cardiac impairment and loss of stamina. This is going to improve all those things. Also, when we look in the liver. Recent research as of 2025 in a mouse model of progressive fatty liver disease demonstrated that ATX converts significant liver and metabolic benefits. So in the study, they were fed a choline deficient high fat diet to induce steatotic liver diseases and then treated with ATx at one milligram per gram

18:20 for several weeks and their liver markers improved. Then when we look at kidney protection, So, beyond other organs, ATX has shown protective effects in acute kidney injury models. So mice were pre-treated with ATx before being given a nephrotoxic dose of cisplatin. millimoles in ATX versus 0.05 in controls, which was a 60% reduction. And then kidney damage markers were dramatically lower with ATx and renal NGAL levels were about one third of untreated.

18:56 So in kidney tubule cell structures, AT x virtually abolished cisplatin induced cell death preserving cell viability. So it suggests that ATX could be a novel kidney protective agent in ischemic or toxic AKI settings, which is pretty cool as well. If you look at kidney health, I know that's one of the things that I get asked all the time is what can I do to improve my kidneys? So when we look the research areas, we have vascular disease. One study reported that prevented pathological changes in abnormal aortic aneurysm model via AMPK activation.

19:27 We look at neurodegeneration. That's one thing I haven't mentioned yet, but when you look all these mechanisms, we obviously are going to have brain benefits. So it's used to rescue neurons from metabolic stress, pre-treatment with O3O4 aka ATX, restored mitophagy and prevented neuronal damage in vitro. And also we have mitochondrial health, so its benefits might extend to protecting neurons via the AMPK-ULK1 mediated mitopathy pathway. suggesting a possible role in neurodegenerative disease models. So overall, all that fancy word, chemical speak aside, preclinical evidence of ATX spans multiple organ systems, not just for fat loss,

20:04 which is pretty cool because we're getting all these benefits as a result. Now let's look at the human trial. So we had the first clinical trial was a 28-day randomized placebo control of phase 2A in patients with type 2 diabetes on metformin as well. They received 1,000 milligrams of ATX once daily as an oral suspension. What we saw was 11 milligrams per deciliter glucose reduction. In patients, with adequately controlled baseline glucose, it achieved a significant reduction in fasting plasma glucose compared to placebo. It also improved their insulin resistant index and caused a significant drop by day 28. We also saw a reduction in blood pressure.

20:40 So there was a statistically significant reduction and diastolic blood and a trend to lower systolic pressure by Day 28 and again, when we talk about some of these fat loss things, that's so cool for me to see because a lot of things are increasing our blood, at least in the short term, if we're talking about like stimulant based things. It also produced a significant increase in peripheral microvascular perfusion and patient skeletal muscle as measured by MRI profusion scans of the calf. So pretty cool. And what is even cooler to me is because we can find a lot of stuff with benefit.

21:11 We look at a safety profile. It appeared safe and well tolerated in the 28-day diabetes trial, so no safety signals or serious adverse events reported, no difference for placebo and reported side effects, and no hypogastemia was observed. And again, not to say anything against GLP-1s because they're amazing, but you don't see that in 28 days on a GLp-one. You're going to have a lot of side effects, especially in a dose dependent manner. So in early trials with exposure up to one month, ATX had no major safety issues and was safe and well tolerated. And unlike some prior AMPK activators, it has shown no cardiac hypertrophy or other concerning effects in animal or human studies.

21:46 So this leads to further clinical development. So building on the diabetes trial, ATX is now being tested in metabolic syndrome and obesity. Amplifier Therapeutics launched a phase 1B trial in 2023, targeting patients with general metabolic dysfunction, not just type 2 diabetes. By 2025, it's progressed to phase one and two trials for obesity, It's being tested alone and in combination with semaglutide, like I said, which if you're in the research world, you are already doing that. If you like myself. And then phase 1B and 2 study in obese individuals is anticipated to measure weight loss, body composition changes, and metabolic health markers.

22:18 I think it'll be really cool to see maybe pairing this with something like a semoglutaride. Does it reduce the muscle loss? Obviously you would pair it with retitrutide if have a brain and you in research, but I now understand like they're doing this. in the context of something like semen glutide that's already well studied and they have super cheap access to. So, while human weight loss results are pending, preclinical data suggests it can induce rapid, sustainable, healthy weight-loss and fat reduction without losing muscle, and I think that is the key. When we look at dosage, like I talked about in that study, they were using 1,000 milligrams.

22:49 Now, to be perfectly frank, I haven't gone that high because I have not felt the need to, And again, i'm not trying to lose a ton of weight or get down to like four or five percent body fat. I've gotten great results at 300 to 400 milligrams per day. Typically, if you're in the research market right now, you are seeing capsules sold at a hundred milligrams a day, I have heard people would experience great result at 100 to 200 milligrams. Right now the thing is just not a lot of people know about this. Hopefully this video helps get more awareness around this product and get people using it. I think it's a great thing. You know, people ask me all the time like what can I use when I'm cycling off a GLP-1 or if I want to stay on a lower dosage of a glp-one.

23:24 Well, Here you go. So great results at 300 to 400 milligrams per day for me. Now, if we were doing the trials, we could go up to 1000 milligrams. And again, I would have zero safety concerns around going that high. I just am always around the minimum effective dose, and I've seen great result of that. But knowing this in the studies, at some point I will probably try going at high And I know for people right now too, that's also like cost consideration thing, because again, this is one of the most, if it's not the expensive thing that we source, it is the one most expensive things that source.

23:54 Again, I'm not saying that to say like, whoa, is me. I am just saying, That's the reality of supply chain right. So in order to get more of this product in people's hands, as kind of evolution we have to go through. We're just doing it a lot smaller scale when you look at kind of like what big pharma charges and the ridiculous rates that we charge, we're trying to do this at a smaller scales so we can actually get these things to be affordable for people. So we look a benefits of mailbox disease. We have better glucose control, reduced insulin burden. And what is really cool is unlike insulin or insulin, so Cretogox ATX does not increase insulin levels.

24:24 It actually reduces hyperinsulinemia by making the body more responsive to insulin. that we're already making. And it addresses cardiovascular risk factors by decreasing blood pressure and improving microvascular perfusion in diabetic patients. So we have a multifaceted benefit by simultaneously targeting hyperglycemia, insulin resistance, dyslipidemia, and hypertension. It's going to be highly relevant to treating and curing metabolic syndrome. We can look at this. I think what's cool about this too is we've got sustainable fat loss. By activating AMPK in fat and muscle, it stimulates the burning of stored fat, inhibits new fat storage.

24:59 Listen to what I said, inhibits new fat storage, leading to reductions in visceral and subcutaneous fat. So in mice on ATX, body fat content drops to near normal despite continued high calorie diets indicating a robust fat burning effect. Now, will that be the same for humans or are you going to continue to eat McDonald's and Bojangles? And not put on weight? Probably not. But I will say if you are on the path of doing things right, this probably gives you a little bit more flexibility in what you're going to be doing. So unlike some weight loss drugs that can cause muscle loss fatigue, it improves exercise capacity and muscle performance.

25:31 I can attest to that. My cardio, granted, I've been testing out a bunch of different stuff lately by regulators and other fat burning things, but man, when you were on this stuff right before cardio it is almost impossible to get tired. We also have the cardiovascular benefits of better blood pressure, better cardiac function, because of increased cardiac stroke volume and efficiency, essentially improving the heart's pumping capacity. We have tissue perfusion by dilating micro vessels, we improve blood flow to muscles and other organs. And we have vascular protection, so early research suggests that it might inhibit vascular inflammation and remodeling, potentially helping maintain vacular elasticity.

26:05 We also have the liver benefits, so it may become a therapy for fatty liver disease. We have protection of the pancreas, its ability to reduce Isola amyloid and support beta cell health. It could help with pancreatic protection, and we also can have kidney protection based on some of those mouse studies. We also have neurodegeneration, so by activating AMPK, it can help clear toxic protein aggregates, which we know are responsible for Alzheimer's, and autophagy induction via AMP can reduce amyloid beta and tau accumulation in Alzheimer models.

26:35 And for someone like myself that probably has a lot of that gray matter in my brain, hopefully less because of peptides, because I played football for 16 seasons. I can't remember how many seasons I've played, right? Because I have so much head trauma. No. I'm just kidding. But I think for anyone that has experienced concussions, hitting this pathway could also be very, very beneficial. Study demonstrated that O3O4 can reduce mitochondrial and autophagic, or excuse me, not reduce, rescue mitochondria and autophasic function in neurons under stress. And also we just have healthy aging.

27:06 So it's strongly supported by animal studies. Treated age mice had improved physical performance and metabolic health and improved health span. Again, we look at this as something that improves health spam and also just to reiterate, very, few, if none at all, safety considerations So what I would do is if you are taking this, just monitor your blood pressure, metabolic panels, and things related to those metabolic panel, but there was no harm in anything that we've seen in humans or animals so far.

27:36 So just to sum up, we have exercise in a bottle with AMPK, all those things we talked about today. really are exercise in a bottle. There's proven benefits. This is statistical at this point. We have tons of statistical data and it is in the works right now. Eventually this will probably become an approved drug based on everything that we see. And then there's going to be a ton of different applications, not just for fat loss, but we have, I think specifically for the brain, the liver, pancreas and the kidneys, a lot of really, really good benefits that can use to help heal some of those chronic issues going on with those organs.

28:08 So that's it for those slides. And that is my comprehensive overview of ATX 304 aka OS 01 aka 03 04, whatever you fancy to call it. So really, really something that I think is amazing that everyone can benefit from. My only desire and goal is to get this more out in the public because I've seen amazing benefits in myself. Again, way better cardio performance, obviously really good fat loss. and just overall better health without some of the side effects that we see in GLP-1.

28:38 So I recommend giving it a try, do as I will, but I appreciate each and every one of you guys. Thank you, guys, so much for all the amazing and overwhelming support. The only issue I have right now is there's so many information to get out and not enough time in the day for me right. Thank you guys for the support. I probably will not be having two videos a week going forward just because of my schedule for next two months, I'm going to have at least one video per week. So stay tuned for that. Promise I'll be doing my best to get those out and make sure that the content is consistent with some of the new information we have out there.

29:08 Thank You guys so much. From the bottom of my heart, it is a dream come true to get to do what I do. And the only reason I get do that is because of you guys. So really, this is about the community. It is making the world a healthier place, point blank period. The fact that I can have a part in that, is the most humbling and amazing thing for me. From bottom my hearts, thank you so much. Whatever form or fashion it, you support the channel. Like, comment, subscribe. sign up for the email list, buy the products, all those things. We are truly a movement that is changing the world and there's so much more room to grow.

29:39 And I'm excited to see where all this stuff goes. So thank you so and I will talk to you in the next one. Peace.